A síndrome de Legg-Calvé-Perthes ou doença de Perthes é uma doença degenerativa da articulação da anca (quadril em português brasileiro) em que ocorre. El absceso del psoas es una enfermedad infrecuente en niños. Puede tener una en pediatría es primario; sin embargo, en ocasiones, puede ser de origen secundario y médula espinal, enfermedad de. Legg-Calvé-Perthes, entre otros Download Citation on ResearchGate | Enfermedad de Legg Calvé Perthes (LCP) en varios aspectos de la enfermedad de Legg Calvé Perthes y la evolución de caderas de August · Anales espanoles de pediatria.
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Espectrophotometric solid-phase tissue plasminogen activator activity assay Sofia-tPA for high-fibrin-affinity tissue plasminogen activators. Changes in coagulation-fibrinolysis system in Legg-Perthes disease: J Biol Chem,pp. Pediatr Res, 35pp. Clin Othop,pp. Severe thrombotic disease in a young man with bone marrow and skeletal changes and with a high content of an inhibitor in the fibrinolytic system.
Thromb Haemost, 71pp.
Síndrome de Legg-Calvé-Perthes
Familial hypofibrinolysis and venous thrombosis. Association of idiopathic venous thromboembolism with single point-mutation at Arg of factor.
Non traumatic osteonecrosis of the femoral head: Familial idiopathic oeteonecrosis mediated by familial hypofibrinolysis witn high levels of plasminogen activator inhibitor.
High risk of thrombosis in patients homozigous for factor V Leiden activated protein C resistance. Continuing navigation will be considered as acceptance of this use.
Thromb Haemost, 78pp. Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Thromb Res, 11pp. Special laboratory evaluation of coagulation. Br J Hematol, 71pp. Blood, 87pp. Protein C and S deficiency, thrombofilia and hypofibrinolysis: To improve our services and products, we use “cookies” own or third parties authorized to show advertising related to client preferences through the analyses of navigation customer behavior.
It has been suggested that the cause of Perthes diseasemay be intravascular thrombosis induced by a potentialcongenital hemostatic disorder leading to conditions ofthrombophilia or hyperfibrinolysis. Relation of altered hemostasis to etiology. Am J Hematol, 45pp. Pathophysiology of osteonecrosis of the jaw: Patients and method Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
Blood, 85pp. Complete study of hemostasis with coagulation and antithrombinfactors as well as study of thrombophilia andfibrinolysis in these patients could determine the prevalenceof hemostasis and fibrinolysis in this group of patients.
Blood, 80pp. Clin Chem, 32pp. Am J Hematol, 48pp. Are you a health professional able to prescribe or dispense drugs? Nature,pp. Anticoagulant protein C pathway defective in majority of thromboembolic patients. J Clin Invest, 94enfermedwd. Am J Clin Pathol, 94pp.
Enfermedad de Legg-Calve-Perthes | HCA Healthcare
Blood, 84pp. Variability of thrombosis among homozigous siblings with resistance to activated protein C due to an Arg-Gln mutaion in gen for factor V. Results Alterations in hemostasis and thrombosis were detectedin one patient who had moderate-to-light protein Sdeficiency. Clin Orthop,pp. Aguirre Canyadell aI. The remaining patients were considered withinthe normal range when age was taken into account.
Conclusions Epidemiological and laboratory data from this group ofpediatric patients and from the control group do not supportthe hypothesis that a thrombogenesis defect couldbe the underlying cause enfermedqd avascular necrosis of the hipjoint. N Engl J Med,pp. Mutation in blood coagulation factor V associated with resistance to activated protein C.
Thromb Haemost, 62pp. Resistance to activated protein C and Legg-Perthes disease. Idiopathic osteonecrosis, hypofibrinolysis, high plasminogen activator inhibitor, high Lp aand therapy with stanozolol. Association of antithrombotic factor deficiencies and hy-pofibrinolysis with Legg-Perthes disease.
Si continua navegando, consideramos que acepta su uso. Blood collection in strongly acid citrate anticoagulant used in a study of dietary influence of basal Tpa activity. Activated protein C resistance as an additional risk factor for thrombosis enfermedae protein C deficient families. Antithrombin heparin cofactor assay with new chromogenic substrates.
Decreased fibrynolitic potential in patients with idio-pathic avascular necrosis and transient osteoporosis of the hip. J Med,pp. Perthes’ disease and the relevance of thrombophilia.